Originally posted at newsroom.ucla.edu
By Mark Wheeler, UCLA Newsroom, November 15, 2012
Last March, researchers at UCLA reported the development of a molecular compound called CLR01 that prevented toxic proteins associated with Parkinson’s disease from binding together and killing the brain’s neurons.
Building on those findings, they have now turned their attention to Alzheimer’s disease, which is thought to be caused by a similar toxic aggregation or clumping, but with different proteins, especially amyloid-beta and tau.
Read the full article at newsroom.ucla.edu.